| Health: |
|
| A ridgeback is generally a very healthy and sturdy dog. It has relatively few |
| genetic diseases compared to other breeds. An average life span of a |
| ridgeback is 10 to 12 years. |
|
| Genetic (hereditary) diseases: |
|
| Degenerative myelopathy (DM) - is a hereditary, genetic disease that |
| usually onsets around the age of eight years and leads to paralysis of the |
| body. The first symptoms are weakness of the hind legs and hind part of |
| the body, tremors, coordination issues. The illness is progressive and the |
| dog gradually loses control of the entire body. In the final stages paralysis |
| occurs, as well as inability to swallow, heart and lung failure. There is no |
| cure or treatment for this disease, but luckily there is a genetic test thanks |
| to which transmisson of the disease to progeny can be successfully |
| avoided. |
|
| A dog can have one of the following genotypes: |
|
| 1. N/N - the dog has two normal copies of the gene, i.e. it does not have a |
| mutation that causes the disease. The dog is healthy, cannot develop the |
| disease, it is not a carrier of the disease and therefore cannot pass it on to |
| its progeny. The progeny of the N/N dog can never be affected by DM. |
|
| 2. N/A - the dog has one normal copy of the gene and one copy of the |
| mutated gene. Such dog cannot develop the disease but it is a carrier of |
| the mutated gene which causes the disease and can pass it on to its |
| progeny. |
|
| 3. A/A - the dog has two copies of the mutated gene. Such dog will be |
| affected by degenerative myelopathy and will inevitably pass on the |
| mutated gene to its progeny. |
|
| The inheritance pattern is as follows: |
|
| 1. If both parents are N/N, i.e. they are not carriers, all puppies will be N/N, |
| i.e. they will be healthy and will not be carriers. |
|
| 2. If one parent is N/N (not a carrier), and the other is N/A (a carrier), all |
| puppies will be healthy (not affected) but all of them will have a 50% |
| chance not to be carriers (N/N) and 50% chance to be carriers (N/A). |
|
| 3. If both parents are carriers (N/A), all puppies will have a 25% chance of |
| being healthy and not carriers (N/N), 25% chance of being affected by the |
| disease (A/A), and 50% chance of being healthy (not affected) but at the |
| same time carriers (N/A). |
|
| 4. If one parent is not a carrier (N/N), and the other is affected (A/A), all the |
| puppies will be carriers but non of them will be affected. |
|
| 5. If one parent is a carrier (N/A) and the other is affected (A/A), all the |
| puppies will have a 50% chance of being healthy but at the same time |
| carriers of the diease (N/A), and 50% chance of being affected by the |
| illness (A/A). |
|
| 6. If both parents are affected (A/A), all the puppies will be affected (A/A). |
|
|
|
| Juvenile myoclonic epilepsy (JME) - is a genetic, hereditary disease |
| which unlike classic epilepsy appear at a young age - between six weeks |
| and 18 months of age. It is characterized by so called myoclonic |
| movements, i.e. jerking of the head and the whole body. The myoclonic |
| spasms or jerks usually occur when the dog is resting or is relaxed, but |
| the seizures can also be caused by bright light. Unlike with the classic |
| epilepsy, there is no loss of consciousness, salivation, foaming etc. |
| Intensity and frequency of seizures vary from dog to dog. Those with |
| milder symptoms have seizures less often and the myoclonic jerks are not |
| as strong, while those with severe symptoms have seizures very often |
| (several times a day) and the jerks are so strong that a dog awakens from |
| sleep, falls down if sitting or standing, cannot normally eat, drink etc. |
| Sadly dogs with severe symptoms have a very poor quality of life and |
| owners usually decide to have them euthanized. Fortunately, scientists |
| have developed a test which helps responsible breeders remove affected |
| and carrier dogs from breeding plans. A dog can have one of the |
| following genotypes: |
|
| 1. N/N - the dog has two normal copies of the gene, i.e. there is no |
| mutation that causes the disease. The dog is healthy, it cannot develop |
| the disease and it cannot pass it on to its progeny. The progeny of the |
| N/N dog can never be affected by JME. |
|
| 2. N/A - the dog has one normal copy of the gene and one copy of the |
| mutated gene. The dog cannot develop JME but it is a carrier of |
| the mutated gene which causes the disease and it can pass it on to its |
| progeny. |
|
| 3. A/A - the dog has two copies of the mutated gene. Such dog will |
| be affected by JME and it will always, without an exception pass on the |
| mutated gene to its progeny. |
|
| The inheritance pattern is as follows: |
|
| 1. If both parents are N/N, i.e. they are not carriers, all the puppies will be |
| N/N, i.e. they will be healthy and will not be carriers. |
|
| 2. If one parent is N/N (not a carrier) and the other is N/A (carrier), all of |
| the puppies will be healhy (not affected by JME) but each of them will |
| have a 50% chance not to be a carrier (N/N) and 50% chance to be a |
| carrier (N/A). |
|
| 3. If both parents are carriers (N/A), all puppies have a 25% chance of |
| being healthy and not carriers (N/N), 25% chance of being affected by the |
| disease (A/A) and 50% chance of being healthy (not affected) but being |
| carriers of the disease at the same time (N/A). |
|
| 4. If one parent is not a carrier (N/N) and the other is affected (A/A), all |
| the puppies will be carriers (N/A) but none of them will be affected. |
|
| 5. If one parent is a carrier (N/A) and the other is affected (A/A), all the |
| puppies will have a 50% chance of being healthy but carriers at the same |
| time (N/A) and 50% chance of being affected by the disease (A/A). |
|
| 6. If both parents are affected (A/A), all the puppies will be affected (A/A). |
|
|
| Hemophilia B (factor IX deficiency) - is a genetic, hereditary disease. |
| Affected animals have a factor IX deficiency. Factor IX (F9) is in charge of |
| blood clotting. Due to the disease, the blood cannot clot properly, which |
| leads to excessive bleeding, even bleeding to death is possible. Bleeding |
| can be caused by injury, trauma or surgery (just like in healthy individuals), |
| but sponataneous bleeding in the internal organs is possible in the |
| affected individuals. Bleeding is not heavier or faster in the affected |
| individuals, it rather lasts too long and cannot be stopped. |
| Depending on sex, each dog can have one of the following genotypes: |
|
| FEMALES |
| 1. Xn/Xn - the dog has two normal copies of the gene, i.e. there is no |
| mutation that causes the disease. The dog is healthy, she cannot be |
| affected, she is not a carrier, and cannot pass it on to her progeny. |
|
| 2. Xp/Xn - the dog has one normal copy of the gene and one copy of the |
| mutated gene. The individual is healthy, but she is a carrier of hemophilia, |
| and she will pass it on to her progeny. 50% of her male progeny is at risk |
| of being affected by hemophilia B. 50% of her female progeny is at risk of |
| being a carrier of the disease. |
|
| 3. Xp/Xp - the dog has two copies of the mutated gene. This dog is sick |
| (affected by hemophilia B), and she will pass on the disease to her offspring |
| without an exception. All of her male progeny (100%) will be affected, and |
| all of her female progeny (100%) will be carriers of the disease. |
|
| MALES |
| 1. Xn/Y - the dog has a normal copy of the X gene. He is healthy, cannot |
| be affected and therefore cannot pass it on to his progeny. |
|
| 2. Xp/Y - the dog has a mutated copy of the X gene. He is affected and |
| he will pass on mutated X gene to all of his female progeny without an |
| exception, i.e. all of his female progeny (100%) will be carriers of |
| hemophilia B. |
|
| The inheritance pattern is as follows: |
|
| 1. Female (Xn/Xn) and male (Xn/Y) - both parents are healthy and are not |
| carriers of hemophilia B, and therefore produce healthy offspring. All |
| progeny is healthy and not carriers. |
|
| 2. Female (Xn/Xn) and male (Xp/Y) - as the female is healthy and not a |
| carrier, all male progeny will be healthy and will not be carriers. As the male |
| is affected, all female offsprings will be carriers of the disease. |
|
| 3. Female (Xp/Xn) and male (Xn/Y) - as the female is a carrier, 50% of all |
| male progeny is at risk of being affected and being a carrier, and 50% of |
| all male progeny has a chance of being unaffected and therefore not a |
| carrier. 50% of all female progeny is statistically at risk of being a carrier |
| of the disease (Xp/Xn), but as the male is healthy none of the female |
| progeny will be affected. |
|
| 4. Female (Xp/Xn) and male (Xp/Y) - as the female is a carrier and the male |
| is affected, all female puppies will be carriers, and 50% of those female |
| puppies is at risk of being affected (Xp/Xp). 50% of male puppies is at |
| risk of being affected (Xp/Y), and 50% of male progeny has a chance of |
| being unaffected (Xn/Y). |
|
| 5. Female (Xp/Xp) and male (Xn/Y) - as the female is affected, all male |
| progeny will be affected (Xp/Y). But thanks to the healthy male, none of |
| the female progeny will be affected, although each female puppy will be |
| a carrier of the disease (Xp/Xn). |
|
| 6. Female (Xp/Xp) and male (Xp/Y) - as both parents are affected, all of |
| their progeny will be affected by hemophilia B - males will be Xp/Y and |
| females will be Xp/Xp. |
|
|
| Early Onset Adult Deafness (EOAD) - is a genetic, hereditary disease. |
| It is characterized by the hearing loss between the age of one and two, |
| and it is therefore different from the deafness puppies are born with and |
| old-age deafness. Affected individuals lose hearing completely and the |
| condition cannot be treated or reversed. |
|
| Each dog can have one of the following genotypes: |
|
| 1. N/N - the individual has two normal copies of the gene, it will not lose |
| its hearing, it is not a carrier of the disease and therefore cannot pass it on |
| to its progeny. |
|
| 2. P/N - the dog has one normal copy of the gene, and one copy of the |
| mutated gene. The individual itself is healthy, it cannot lose its hearing, but |
| it is a carrier of the disease and will pass on the mutated gene on to its |
| offspring. |
|
| 3. P/P - the dog has two copies of the mutated gene. The dog is affected, |
| i.e. will develop the disease (hearing loss), and it will pass on the mutated |
| gene to its progeny without an exception. |
|
| The inheritance pattern is as follows: |
|
| 1. If both parents are healthy and not carriers (N/N), all of their puppies |
| will be healthy and will not be carriers of the disease (N/N). |
|
| 2. If one parent is healthy and not a carrier (N/N), and the other is a carrier |
| (P/N), none of their progeny will be affected but each will have a 50% |
| chance of not being a carrier (N/N), and 50% chance of being a carrier (P/N). |
|
| 3. If both parents are carriers (P/N), all of their puppies have a 25% chance |
| of being healthy and not being carriers (N/N), 25% chance of being affected |
| (P/P), and 50% chance of not being affected but being carriers (P/N). |
|
| 4. If one parent is healty and not a carrier (N/N), and the other is affected |
| (P/P), all of their progeny will be carriers but will not be affected by the |
| disease (P/N). |
|
| 5. If one parent is a carrier (P/N), and the other is affected (P/P), all of their |
| progeny has a 50% chance of not being affected and being carriers (P/N) |
| and a 50% chance of being affected (P/P). |
|
| 6. If both parents are affected (P/P), all of their progeny (100%) will be |
| affected (P/P). |
|
|
| Hip dysplasia (HD) - is a genetic, congenital condition. It is a |
| developmental deformity of the hip. The ball of the femur is loose in the |
| socket due to weak ligaments, tendons and muscles. Due to the improper |
| position of the joint, the cartilage and the bone wear and tear, causing |
| pain, lameness and difficulty moving. Hip dysplasia can only be surgically |
| treated. |
|
| A certified radiographic examination (x-rays) which is used to score dog's |
| hips should be done once a dog has finished its growth and development |
| (for ridgebacks ideally at or after the age of 18 months). |
| FCI hip classification is as follows: |
|
| A - a normal hip without signs of dysplasia. |
| B - an almost normal hip (transition from normal toward dysplastic hip). |
| C - mild hip dysplasia. |
| D - moderate hip dysplasia. |
| E - severe hip dysplasia. |
|
| Each hip is scored separately so the result is displayed as A/A, A/B, B/B etc. |
|
| FCI allows breeding of dogs with A, B and C hips, while dogs with D and E |
| hips must not be used in breeding. Naturally, a breeder should for |
| breeding use dogs with the best possible hips (A hips ideally), and avoid |
| those with poor(er) hips (B and C) whenever possible. |
|
|
| Elbow dysplasia (ED) - is a genetic, congenital condition. It is |
| represented by improper and disproportional development of bone and |
| cartilage components of the elbow. These lead to mechanical and |
| inflamatory changes in the joint which cause pain and lead to degenerative |
| changes on the cartilage. The affected elbow is painful and the joint's |
| movement is limited and improper. Just like hip dysplasia, elbow dysplasia |
| can only be surgically treated. |
|
| A certified radiographic examination (x-rays) which is used to score dog's |
| elbows should be performed once dog's growth and development has |
| completed (for ridgebacks ideally at or after the age of 18 months). |
| FCI classification of elbows is as follows: |
|
| 0 - a normal elbow without signs of dysplasia. |
| 1 - mild dysplasia (transition from normal to dysplastic). |
| 2 - moderate dysplasia. |
| 3 - severe dysplasia. |
|
| Each elbow is scored separately and results are presented as 0/0, 1/1, 2/3 |
| etc. |
|
| Only healthy dogs with 0/0 elbows should be used for breeding. |
|
|
| Other illnesses: |
|
| Bloat - Just like other breeds with similar built (large dogs with deep chest) |
| a Rhodesian ridgeback is prone to bloat (torsion or twisting of stomach). |
| This is a serious, potentially fatal condition that requires immediate |
| surgery. The bloar occurs when a dog's stomach twists around its own |
| axis so the amount of accumulated fluid and gases cannot be reduced by |
| burping or vomiting. When a stomach twists the blood circulation in the |
| stomach and small intestine gets cut off. The bloat usually happens when |
| a dog overeats, gulps down food and air fast, drinks large amount of |
| water immediately after the meal or when a dog jumps or runs after the |
| meal. Symptoms develop very quickly - the dog tries to vomit but cannot |
| throw up the content from the stomach, he is agitated, the stomach swells |
| up, mucous membranes become pale, the dog becomes weak and the |
heart rate increases. If left untreated, bloat can quickly cause shock and
|
| death. |
|
| This is why it is necessary to take special care while feeding your ridgeback, |
| i.e. prevention. The dog should be given several smaller meals during the |
| day (at least two) always AFTER a walk or exercise. Do not allow your dog |
| to overeat, run or jump at least three hours after the meal. Do not let him |
| drink water immediately after eating and wait at least an hour before you |
let him drink.
|
|
|
| Dermoid Sinus - This is a hereditary, genetic disease. A dermoid sinus is an |
| abscess (or a tube) that runs from the surface of the skin down to the |
| spinal cord. The tube fills with hair and skin debris and fluids accumulate in |
| it. It leads to infection that eventually reaches the spinal cord and leads to |
| a painful death. The affected puppies have a small lump (usually on their |
| necks and backs) and a small hole is visible when the hair is shaved off. The |
| only treatment is surgery but it is usually unsuccessful and puppies with |
| dermoid sinus are usually put to sleep. Thanks to proper selection and |
| breeding, dermoid sinus is not very common anymore. |